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In pathophysiological conditions, PKD-mediated phosphorylation of Ser637 has been demonstrated to enhance mitochondrial fragmentation (Serasinghe and Chipuk, 2017), highlighting the need for systematic elucidation of its spatiotemporal regulatory mechanisms and precise molecular determinants governing mitochondrial fission dynamics

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By methylating and removing nicotinamide, NNMT effectively drains NAD+ precursor availability, leading to reduced intracellular NAD+ levels